Transaldolase deficiency influences the pentose phosphate pathway, mitochondrial homoeostasis and apoptosis signal processing.

Citations & References

  • Authors: Qian Y, Banerjee S, Grossman CE, Amidon W, Nagy G, Barcza M, Niland B, Karp DR, Middleton FA, Banki K, Perl A,
  • Journal: Biochem J (2008) 415:123-134
  • ID: PN66913
1754754f941af7bb9c557995d8d24c5b_Citations

Caspase-cleaved tau expression induces mitochondrial dysfunction in immortalized cortical neurons: implications for the pathogenesis of Alzheimer disease.

Citations & References

  • Authors: Quintanilla RA, Matthews-Roberson TA, Dolan PJ, Johnson GV,
  • Journal: J Biol Chem (2009) 284:18754-18766
  • ID: PN67635
d83eee8a7cb91234d1fa3e76d2be139f_Citations

The Psi(m) depolarization that accompanies mitochondrial Ca2+ uptake is greater in mutant SOD1 than in wild-type mouse motor terminals.

Citations & References

  • Authors: Nguyen KT, García-Chacón LE, Barrett JN, Barrett EF, David G,
  • Journal: Proc Natl Acad Sci U S A (2009) 106:2007-2011
  • ID: PN67529
e557c8251ac0552a147af7f07bebd5c5_Citations

PINK1-associated Parkinson′s disease is caused by neuronal vulnerability to calcium-induced cell death.

Citations & References

  • Authors: Gandhi S, Wood-Kaczmar A, Yao Z, Plun-Favreau H, Deas E, Klupsch K, Downward J, Latchman DS, Tabrizi SJ, Wood NW, Duchen MR, Abramov AY,
  • Journal: Mol Cell (2009) 33:627-638
  • ID: PN67531
286b0a5bae712b5c143bcc000151aac4_Citations